This section examines the relationship between smoking uptake and the use of other substances among young people. It outlines patterns of concurrent and sequential use of tobacco with alcohol, cannabis and other drugs, and considers how these behaviours interact to influence initiation, progression and dependence. The section also discusses individual, social and biological factors that may help explain the co-occurrence of tobacco use with other forms of substance use.
As noted in Section 5.5, tobacco use in adolescence is associated with other risky behaviours, including other drug use. Australian and international research consistently shows that people who smoke are more likely than those who do not smoke to drink alcohol (including frequent binge drinking),1 use cannabis and other illicit drugs2,3,4 and sniff glue or petrol.5
Concurrent use of tobacco and alcohol appears to affect the quantity of consumption. For example, in research among US college students who were both smoked and drank alcohol, (mean age 20 years) students drank more while smoking and smoked three times as many cigarettes, on average, during drinking episodes.6 Smoking while drinking was more likely when students were with others at a party or a bar, while increased stress between assessments predicted a greater likelihood of smoking while drinking.6 High rates of drinking common among young adults may contribute to the transition from less-than-daily to daily cigarette smoking, for example by altering the subjective effects of smoking such as satisfaction and enjoyment; preliminary research among young adults found that alcohol consumption was associated with a reduction in some of the negative effects associated with smoking and with an increase in subsequent smoking levels.7
Evidence from a US longitudinal study among young adults followed over three years suggests that even occasional smoking is predictive of hazardous drinking and alcohol use disorders, after controlling for baseline drinking behaviours.8 Concurrent tobacco and alcohol use among young people aged 15 years has been associated with greater nicotine dependence, significantly higher levels of consumption and more excessive use of alcohol, earlier drinking uptake, and more cannabis use, compared with those who consumed only alcohol. Similarly, compared with those who used tobacco only, concurrent users reported higher nicotine dependence and more cannabis use.9 The severity of nicotine dependence has also been found to significantly predict alcohol-related problems among adolescents who smoke in a smoking cessation clinical trial.10
In Australia, the average age at which young people had their first cigarette (16.6 years) or full glass of alcohol (16.1 years) was very similar, at about 16 years of age. Initiation of cannabis use among young people occurred at an average age of about 19.1 years, and first use of ecstasy occurred at an average age of 21.7 years. Among Australian secondary school children aged 12–17 years who reported having used other substances in the previous 12 months, 42% who had taken ecstasy, 39% of cannabis users, 36% who had used amphetamines,38% who had used hallucinogens and 13% who had used tranquilisers reported also using tobacco at the same time.11
A survey among more than 29,000 young French people aged 17 years revealed that the likelihood of first initiating tobacco was far greater than the likelihood of initiating cannabis, while the risk of experimentation with cannabis or tobacco was much greater once a student had experimented with one of these substances.12 Research among some groups of US young people has found the association between trajectories of tobacco and cannabis use from adolescence into adulthood to be quite high,13 with some evidence that ‘smoking’ may represent a social construct and an activity domain within which the use of cigarettes or cigars containing tobacco and/or cannabis is somewhat interchangeable.14
In some settings, however, it appears that it is the wish to use cannabis that leads to tobacco addiction, tobacco being used initially as a means of facilitating cannabis smoking.15,16 This pattern of use has been reported among some Aboriginal and Torres Strait Islander populations17 (see Chapter 8, Section 8.11.2). Similarly, Swiss research among young cannabis users found the mixing of tobacco and cannabis in cannabis cigarettes to be very common, especially among daily tobacco users,18,19 with some current and former cannabis users perceiving cannabis to be more natural, less harmful and less addictive than tobacco.18
Cannabis use has been linked with the transition from experimental to daily tobacco smoking in late teenage and early adult years.20 Victorian research found that adolescents and young adults who use cannabis on a regular basis are more likely to progress to tobacco initiation and dependence, even after controlling for obvious potential confounding factors such as peer group and family influences.21 The authors of this study suggest a number of reasons why the behaviours may be interlinked, including the propensity for cannabis and tobacco to be used in combination, biological mechanisms, and nicotine functioning as a way of ameliorating symptoms of withdrawal from cannabis use. Research among Finnish twins suggests that several common genetic influences may be related to tobacco and cannabis (and other illicit drug) use; common and unique environmental factors were also very important, while the direct impact of tobacco smoking uptake explained most of the variation in the initiation of illicit drug use.22
The connection between smoking and the use of other drugs is likely to be due to a combination of individual, social and environmental factors, including temperamental characteristics.23 For example, evidence from The Netherlands suggests young adolescents who report higher levels of hopelessness and sensation seeking are at higher risk for an early onset of substance and poly-substance use (including alcohol, tobacco and cannabis).24
There is also some evidence that the biological changes in the brain caused by early exposure to nicotine may also influence neurological response to other drugs, such as alcohol and cannabis, and other drugs of abuse, leading to a greater risk of ongoing use and addiction.4
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References
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